What brings the theory back? Researchers at the Buck discovered that they could specifically block free radical production in mitochondria. An elegant solution replaces an indiscriminate and messy ...

This is because free radicals, which can cause DNA damage, are produced during ATP synthesis. Mitochondria also lack the same protective mechanisms found in the cell nucleus. However, the majority ...

These free radicals mess with our cellular metabolism, even interfering with our DNA. Our mitochondria, which operate like little factories in our cells, are responsible for burning fuel from food ...

Proponents argue that as the body ages, it accumulates free radicals. This results in more oxidative stress and more damage to cells, or their mitochondria, which contributes to aging. The theory ...

This turns the victimized molecules into free radicals themselves, perpetuating a vicious cycle that can cause cellular damage or even cell death. As mitochondria age, they seem to produce more free ...

In the current study, the scientists show that stimulation of the beta-adrenergic receptors also leads to increased production of free radicals in the mitochondria of the cells, and these then ...

Over time, this area of research went on the shelf as mitochondrial theories of disease and aging fell into disfavor. But research at the Buck offers a new way to deal with free radicals: rather than ...

More information: José Villalaín, Location and interaction of idebenone and mitoquinone in a membrane similar to the inner ...

“ALA helps improve the mitochondrial efficiency of the neurons and provides a cofactor from important cellular enzymes.” An overabundance of free radicals can be the result of internal or ...

A study published on July 20 in the journal Nature reveals that the mitochondria in oocytes tap into a different energy pathway that sidesteps the production of free radicals—chemical molecules ...