Amoebas don’t usually make headlines. But Entamoeba histolytica, a single-celled killer with a taste for human cells, may ...

Entamoeba histolytica occurs in the human body in four ... motile and does not contain bacteria or other food inclusions (4) ...

Serologic testing was positive for Entamoeba histolytica antibodies ... After ingestion of E. histolytica, the cysts hatch into trophozoites within the intestine. The motile trophozoites then ...

Parasites are known to infect human cells through a variety of ingenious mechanisms. Many of them have even evolved sophisticated strategies to evade a host’s immune system by avoiding it entirely.

Scientists have revealed how a parasite that affects millions of people every year evades our immune defenses.

ALA = Amoebic liver abscess; Eh = Entamoeba histolytica; EhCP = E. histolytica cysteine proteinase; Ehserp = E. histolytica serine proteinase; EhSTIRP = Serine threonine isoleucine-rich protein ...

Amebiasis is an infection of the gut caused by a parasite called Entamoeba histolytica ... the wall of the large intestine and forms a cyst, this may cause ulceration and bleeding.

For all subjects, the presence of E. histolytica in the stool was reassessed with the use of the PCR assay 10 and 20 days after the termination of therapy. There was no loss to follow-up.

It has not hitherto been detected in the protozoan, Entamoeba histolytica. In earlier studies, Nakamura 1 reported the absence of casease in certain strains of the amœbæ. It was thought that the ...

The mechanisms whereby certain mouse strains develop persistent intestinal infection with Entamoeba histolytica remain unclear. In this work, we characterized the kinetic pattern of cytokine ...

Entamoeba histolytica infects nearly 50 million people each year and kills around 70,000. It’s a shape-shifting parasite that ...

Amoebiasis caused by Entamoeba histolytica is the second leading parasitic cause of human mortality, mainly affecting children of the developing world. Recent sequencing of the genome of E.